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When I went to the ER to find out what was wrong they said my heart was in afib and it was beating irregular at about 190bpm. They put me on a cartizem drip all night and my heart converted on its own the next night. I am now on cartizem three times a day and have not had any other episodes of afib other than an occasinal flutter. I went to regular doctor and she did ekg said it looked ok but not great and heart rate was a littel high 96bpm but could be from me being so scared that I am going to drop dead.

Cytokines, such as TNF-α, CRP, IL-6, IL-8 and IL-10, are some of the biomarkers found to be elevated in AFib , as are increased populations of neutrophils and lymphocytes proportionately (NLR ratio) in AFib patients compared to those in sinus rhythm (SR) . Thus the “AFib begets AFib” phenomenon continues, but it remains to be determined whether inflammation is the incipient cause, consequence, or both with respect to AFib pathophysiology and thus clinical outcome. Atrial Fibrillation (AFib) is an irregular heart rhythm that affects ~ 1.5% of the population globally (~ 10% adults over 80 years of age), can lead to heart failure or stroke, and is a major determinant in patient quality of life . AFib may be asymptomatic or manifest various symptoms such as palpitations, fatigue, shortness of breath, dizziness, and chest pain .

There also appeared to be a dose-dependent increase in the number of collisions with an apparent two-fold increase in the number of individuals having a collision with only a modest increase in the amount of THC administered (i.e. 4 mg or a 23% increase in THC). Subjective effects were also examined and the study found a significant increase in physical discomfort, physical effort, and lack of energy with the highest dose of THC compared to placebo, although the lowest dose also produced physical discomfort and effort. Although both the lowest THC dose (13 mg) and alcohol (0.05% BAC) appeared to produce driving impairment, there appeared to be differences in subjective effects between THC and alcohol.

Another study demonstrated dose-dependent sex differences in subjective responses to CBD gummies for sale orally administered THCReference 564. In this study, women showed greater subjective effects at the lowest dose (5 mg), whereas men showed greater subjective responses at the highest (15 mg) dose.

In addition, in contrast to the findings obtained for acute pancreatitisReference 1296, tissues isolated from patients with chronic pancreatitis appeared to have decreased levels of both anandamide and 2-AGReference 1297. Activation of CB1 and CB2 receptors in chronic pancreatitis-derived pancreatic stellate cells was also associated with the induction of a quiescent-cell phenotype as well as the downregulation of extracellular matrix protein production and inflammatory cytokine productionReference 1297. Injection of anandamide or a CB1 receptor-selective agonist in rats was associated with acute glucose intolerance, whereas administration of a CB1 receptor inverse agonist attenuated this effectReference 1294. In humans, intravenous injection of 6 mg of Δ9-THC to healthy, non-obese, male volunteers was associated with acute impairment of glucose tolerance in response to glucose challenge with no change in plasma insulin levelsReference 1295. In contrast to the effects of acute CB1 receptor agonism (e.g. acute THC exposure), studies examining the effects of chronic cannabis use on body weight and metabolic status in non-clinical populations have found the opposite effects.

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